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MizzouForward Keynote – Synaptosome microRNAs and synapse function in Alzheimer's disease

 

 

January 18th | 11:00am – 12:00pm

572 Bond Life Sciences Center

Join us for Synaptosome microRNAs and synapse function in Alzheimer's disease" a keynote presentation by College of Veterinary Medicine - Biomedical Sciences, MizzouForward faculty candidate, Dr. Subodh Kumar. Dr. Kumar will present on his research for approximately 40-minutes with a 20-minute question and answer session to follow.

 

Dr. Subodh Kumar is currently as Assistant Professor in the Center of Emphasis in Neuroscience, Department of Molecular & Translational Medicine at Texas Tech University Health Science Center, El Paso, TX.  Dr. Kumar completed his B.S. degree in Zoology, Botany, Chemistry and his M.S. degree in Biotechnology (specialization in Molecular Biology) from Choudhary Charan Singh University, Meerut, India.  He earned his Ph.D. in Molecular Genetic and Biotechnology, Post Graduate Institute of Medical Education and Research, Chandigarh (PGIMER), India.

 

Synapses are one of the most important compartments of neurons that deliver signals to adjacent neurons and maintain optimal synaptic functions of the healthy brain. Dysregulation of synapses is the initial impairment that causes synaptic degeneration in Alzheimer’s disease. Neuron-specific microRNAs (miRNAs) are localized at synapse. Synaptic vesicles and synaptosomes are enriched with miRNAs. However, the role of synapse-associated miRNAs in the pathogenesis of Alzheimer’s disease is unknown. The major goal of Dr. Kumar’s research is to investigate the functions of synaptosomal miRNAs in Alzheimer’s disease progression and pathogenesis. The findings from these studies could be groundbreaking, which will allow developing suitable strategies for the treatment of Alzheimer’s disease.

 

You can access Dr. Kumar’s CV via OneDrive here:

 ​Subodh Kumar CV 12-14-22 .pdf icon Subodh Kumar CV 12-14-22 .pdf (University log in required to access)

After the keynote, please provide candidate feedback with our brief survey.

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